Christian Louboutin Greece mgr ufe xhce smc

asbryobvrz · Wysłany: Nie 9:43, 27 Mar 2011

Baiao Lumbrokinase anticoagulation treatment of acute cerebral infarction, fibrinolytic system impact analysis

PT, KPTT change from the original determination of protein and fiber. 2 Results 2.1 million Austrian lumbrokinase after taking extended KPTT, PT no significant changes (Table l ~ 2). Table 1 treatment group and control group change in Table 2 KPTT treatment group and control group,[link widoczny dla zalogowanych], changes of PT units: Linhai City, Zhejiang Province, the Second People's Hospital (Taizhou 317016) Note: P> 005. Chinese medical research and clinical. April 2007. Volume 5. 4 2.2 Some patients (n = 15) n / ll ~ Baiao lumbrokinase fibrinogen decreased after 2 weeks, and this change are obviously significant (Table 3). Table 3 changes in the control group fibrinogen Note:, '<0OI. 3 Discussion normal physiological circumstances, the body of coagulation and anticoagulation system in dynamic equilibrium, so that the blood flow in the vessel to maintain the state. It is reported that the blood of patients with ischemic stroke in a high coagulation, low fibrinolytic state J. The pay would be a hundred Austrian lumbrokinase enzyme, with direct thrombolytic and activation of fibrinolysis, platelet aggregation, expansion and other capillary effect. The mechanism mainly two: the first is the role of plasminogen activator; second, and fibrin have a special affinity with the fiber protein binding,[link widoczny dla zalogowanych], so that the rapid degradation of fibrin. Experiments in patients with oral Baiao lumbrokinase KPTT before and after treatment was significantly longer (P <0.05), as KPTT an endogenous coagulation pathway is sensitive to the screening index, which reflects the extension of the various factors of the endogenous coagulation system The reduction in procoagulant activity, which shows Baiao lumbrokinase inhibit endogenous coagulation system. Anticoagulant composition for the existence lumbrokinase, reportedly is a different and independent of heparin on the antithrombin re (AT-m) to play the role of anticoagulant substances J. Secondly, the patients after oral Baiao Lumbrokinase,[link widoczny dla zalogowanych], decreased plasma fibrinogen levels, which can lead to hypercoagulable state, reduced blood flow. Therefore, by reducing the concentration of fibrinogen is the treatment of ischemic cerebrovascular disease strategies. The experimental results confirmed that lumbrokinase lower fibrinogen concentration may be through the activation of plasminogen, so that due to rapid degradation of fibrinogen. In short, a hundred Austrian lumbrokinase fibrinogen can be induced reduced, KPTT extension, it has a number of anti-thrombosis effect of direct factor,[link widoczny dla zalogowanych], but no effect on serum lipids, no lipid-lowering effect. The merger with hyperlipidemia,[link widoczny dla zalogowanych], needs to be treated with other lipid-lowering drugs. In summary, the percentage can be widely used in Austria lumbrokinase prevention of ischemic stroke and prevention of the occurrence of other thrombolytic, anticoagulant therapy after the re-formation of thrombus. 【
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