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yxfbbiedtj · Wysłany: Wto 10:23, 15 Mar 2011

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Idiopathic pulmonary hemosiderosis and evaluation of pulmonary function in patients

Brittle damage function mainly as hypoxemia (I-call back, pI-I> 7.45 were down 3 down} l4 rest were normal pH. Declined), diffuse power brittle obstacles and limited ventilation barriers. The group down in l8 22 l8 back down I-bang for the power failure combined brittle diffusion barriers, PaO I-bang for the decline. Because hyperventilation. PaCO most of the normal, or even 3.4 ~ 6.2 kPa (5.53 soil 0.87kPa), PADO: 6 ~ reduced. 19 down PA-aDO2 showed increased diffusion dysfunction, 510.9kPa (8.14 Soil 1.45 kPa) I fell for the type II respiratory failure (PaO ~ down routine lung function measurement with 2 inverted chip crisp limited ventilation barriers; I 35kPa, P ~ CO, 81,[link widoczny dla zalogowanych],, Pa),[link widoczny dla zalogowanych], I fell only a diffuse dysfunction (PA grapefruit down due to bleeding obstruction,[link widoczny dla zalogowanych], restrictive ventilatory defect on the basis of further consolidation DOz7kPa). 2 down more than normal blood. Obstructive ventilatory defect, is a mixed ventilatory defect. 5 down Strength Qigong II, fragile lungs measured through Qigong: 5 fall in this group did pulmonary function were significantly less brittle force,[link widoczny dla zalogowanych], determination and confirms the results of the pathological mechanism. Determination, 2 down is restrictive. FVC and Mw <71; 3 down help understand the disease of pulmonary function, Pán off after Ru are mixed and guidance. In addition to a restricted type of change outside, FEVo <60%. Pulmonary oxygen therapy. Significant changes in lung function were crisp devaluation prompted extensive disease, illness saddle weight. Strong function were significantly decreased gas. For irritability, tension, faster breathing, heart rate, cough and other early lack of three, by hormones, oxygen and other comprehensive treatment, 9 1 week left in the oxygen back who does not appear cyanotic, and blood gas changes, to be given oxygen therapy . Review of the right arterial blood gas, respiratory failure 7 I-down power combined diffusion barriers in brittle masks, oxygen effect satisfied the first box, to give the CPAP treatment. Returned to normal, there are still 2 down I-respiratory failure and diffusion barriers, suggesting that disease fell by 5 car group satisfied with CPAP efficacy. Delay treatment process more difficult. Through hormones and oxygen therapy combined therapy, our hospital discussed 4O down IPH, 36 improved and discharged down the death 4 down. Analysis of early death of IPH was not trapped rare disease. China from 1960 to 1983 only slightly on the pan to suffocate. The late secondary pulmonary infection. Pediatric cases reported in the treatment of 100 down. Since 1986, our hospital were treated in 40, the dynamic monitoring of lung function for increasing response rates and reduce mortality from IPH cause has not yet finished down clear bases, and alveolar epithelial cells in fat may be crucial. Education and functional abnormalities, immune function brittle obstacles, and access to fresh milk allergy. . . . . . . Jing touch four organophosphorus pesticides causes. Alveolar pathology on