MBT schuhe preise Chronic pulmonary heart disease

xp574088 · Wysłany: Pią 11:30, 25 Mar 2011

Chronic pulmonary heart disease and multiple organ failure Efficacy

Killed, dried, combined with other organ failure have varying amounts of relationship. Table 2. Table 2 The number of deaths associated with other organ failure statistics in Table 2 cP and the MOF, the digestive system failure, higher mortality rate, followed by nervous system complications, and metabolic dysfunction of the urinary system and mortality was low. 3 Discussion acute exacerbation of chronic pulmonary heart disease a higher incidence of MOF, and poor prognosis ¨]. MOF with trauma or surgery is different, the former is slow loss of lung based on the occurrence of heart, lung infection in this group of cases are for the incentive, leading to acute exacerbation of pulmonary heart disease, Gram-negative bacteria produce endotoxins or inflammatory cytokines in addition to direct damage to organ function,[link widoczny dla zalogowanych], but also affect hemodynamics, can lead to organ hypoperfusion, ischemia, hypoxia, 1669 * into the form microthrombi. _ Endotoxin can stimulate the body to produce immune complex deposition in multiple organs of the endothelial cells, the immune response. Transmitter through a number of inflammatory-mediated, the occurrence of systemic inflammatory response syndrome (SIRS), organ damage, causing multiple organ dysfunction syndrome (MODS), further increase will lead to MOF. Pulmonary heart disease with acute exacerbation of the bronchial mucosal hyperemia, edema, bronchospasm, mucus plugging causes such as hypoxia and hypercapnia, hypoxemia increased anaerobic metabolism to tissue cells, ATP, and decreased production of cAMP, leading to organ of energy metabolism . Oxygen and carbon dioxide retention caused by pulmonary artery spasm, increased pulmonary hypertension, increased right ventricular afterload, decreased right ventricular cardiac output, left ventricular preload reduction, combined with the impact of bacterial toxins on the heart to decrease myocardial contractility, cardiac output further reduce the amount of the effective circulating blood volume deficiency, decreased perfusion of tissues and organs, causing MODS, and tissue and organ hypoperfusion that capillary endothelial damage,[link widoczny dla zalogowanych], can cause DIC. As patients were hospital late, so that disease progression, and that one or more organ failure, 1 patient in this group of 6 up to organ failure. Our data show that when the pulmonary heart disease complicated by MOF to the most common metabolic failure, a total of 25 cases,[link widoczny dla zalogowanych], accounting for 36.8% MOF group, 2 patients died, accounting for 12% of metabolic failure. Hypokalemia, metabolic alkalosis failure, low chlorine leading cause of death due to alkali from the curve to the left of oxygen poisoning, oxygen and hemoglobin can not release the close of the vital organs of oxygen increased. Potassium and neuromuscular excitability can reduce a range of signs and symptoms appear, and difficult to correct the metabolic disorder and death; followed by 20 cases to renal failure, accounting for 29.4%, and 1 died of renal failure accounted for 5 .0%. Yi Yin hypoxia and hypercapnia, reflex caused by renal vasoconstriction, decreased renal blood flow, glomerular filtration rate reduction caused by renal failure, severe cases can cause death; pulmonary encephalopathy in 16 cases, 23 .5%, 3 patients died of pulmonary encephalopathy accounted for 16.7%. Inhibited by hypercapnia and cerebral cortex and subcortical drowsiness, lethargy and even coma, while allowing expansion of cerebral vascular and peripheral vascular, cerebral blood flow, brain volume increases, increased intracranial pressure, eventually leading to encephalopathy and shock and death; digestive system failure, 7 cases,[link widoczny dla zalogowanych], 10.3%, 6 died, 85.7% for the digestive system failure, poor prognosis,[link widoczny dla zalogowanych], the mortality rate is very high. The more the number of organ failure and higher mortality rates, in order to reduce mortality, the key is to reduce the number of organ failure and prevention of failure and eliminate the occurrence of metabolic alkalosis. First, we should actively fight infection, expanding the airway, monitoring vital signs, coagulation parameters, potassium, sodium, chloride and blood gas analysis. Early identification and treatment, and promptly correct the oxygen and carbon dioxide retention. I believe that for respiratory acidosis and (or) on behalf of the determination of acid potassium normal when the patient should supply the daily physical requirements 2-3g, renal failure and anuria are exceptions. At the same time make chlorine, after correcting acidosis prevention of hypokalemia, low chlorine alkalosis. Serum potassium and chloride decreased again when the event of potassium chloride is too late. If severe hypoxemia and hypercapnia, the amount should be given oral or intravenous cimetidine infusion routine to prevent gastrointestinal bleeding. In addition to the prevention and control, the need to systemic supportive therapy to maintain the environmental balance and improve immune function and other comprehensive measures to avoid the occurrence of MOF continuity and reduce mortality. 4
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