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Wysłany: Nie 4:27, 06 Mar 2011
Temat postu: tory burch flats ndx gxl bnn whq
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Ⅱ plasma endothelin in patients with diabetes and blood glucose,
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, insulin,
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, triglyceride relationship
Depression: * compared with the same fasting P <001; A comparison with the control group P <0. O1. Animal experiments showed that Insulin stimulates endothelin gene transcription and protein secretion, increased plasma endothelin. The group observed that the 2-hour postprandial insulin diabetic patients, ET than fasting, showed a significant correlation. Control group, insulin, endothelin was increased after the meal compared with fasting, but no significant difference. That diabetic patients with insulin increased, Nei Pisuo secretion increased, higher concentrations of insulin that can stimulate endothelin release in the body, consistent with the foreign reports. In recent years, the postprandial blood lipid status has been getting attention lotus. Postprandial metabolism of glycerol triol as an independent risk factor for coronary heart disease ∽. It was found abroad. When the acute increase in triglycerides. Insulin significantly increased endothelin 【also will be increased significantly. This observation also found in the diabetic group compared with postprandial fasting glycerol glycerol increased postprandial than fasting also increased ET. Was a significant correlation. Postprandial triglyceride in the control group, Nei Pisuo was increased compared with fasting, but suggested no significant difference in the reasons is because the glycerol glycerol can be elevated tank, hyperviscosity, the formation of vascular endothelial cells. To Nei Pisuo release increased. At the same time, glycerol glycerol also co Insulin stimulation of endothelin release. In conclusion, plasma endothelin levels in patients with diabetes, higher than normal, blood glucose, Insulin, triglycerides is to stimulate the secretion of plasma endothelin in patients with diabetes increased the important reasons. The enhanced role of postprandial. Endothelin, a strong pool of vascular function, combined with hyperlipidemia, hyperinsulinemia, and other factors, diabetic patients the incidence of atherosclerosis, promote the development cycle while increasing barriers and forging San vascular lesions. Observed by this group, we believe that diabetic patients should be strictly controlled blood sugar, blood lipids, reduce high Insulin hyperlipidemia to prevent, delay and prevent neurovascular complications of diabetes, the development of
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