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Wysłany: Wto 15:23, 22 Mar 2011
Temat postu: Christian Louboutin Greece ipp mnz clbi reh
Gingival fibromatosis a case report
alfibromatosis), also known as hereditary gingival fibromatosis (hereditarygingivalfibromatosis), also known as familial (familia1) or idiopathic (idiopathic) gingival fibromatosis is a rare disease. The etiology is unknown, and often familial autosomal dominant inheritance,
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, but some patients have no family history. I admitted to hospital in March 1997 a case of full mouth gingival fibromatosis patient, reported as follows. The clinical data of patients 1 female, 23 years old,
Christian Louboutin Greece
, complained of upper and lower gingival enlargement twenty years. No bleeding, no overflow pus, no pain. Found that patients with full mouth at birth with mild gingival hyperplasia with age, gingival swelling gradually increased. Patients denied a similar family, no drugs, food allergies. Examination findings: maxillofacial symmetry, full mouth gingival hyperplasia showed fibrous covering gums, cheek tongue, revealing only after the anterior margin and occlusal, gingival color, subdued, no bleeding tenderness, no overflow pus, ulcers. Treatment: local anesthesia full mouth gum surgery. 2 discussion gingival fibromatosis gingival epithelial tissue thickening is seen, spikes up, deep connective tissue, gums, connective tissue volume increase. Capsule filled with coarse collagen fibers and numerous fibroblasts. The relative reduction of vascular inflammation near the gingival sulcus only see above. Clinical manifestations usually after the eruption of permanent teeth, gums that generally increased, the gums can spread to the whole mouth gingival membrane attached directly to the joint at the gingival. A small number of children in the deciduous teeth of the disease. Gingival hyperplasia normal color, firm, smooth or nodular performance, stippling evident. Easy bleeding, pain, hyperplasia in the maxillary molars, 7 palatal most. In severe cases,
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, the gums cover the entire crown, hinder chewing function, children sometimes sprout difficult. Brucellosis diagnosis based on typical clinical manifestations and positive family history, no family history can not be ruled out. Differential diagnosis: (1) drug-induced gingival hyperplasia. Based on: ① no medication history and family history; ② drugs hyperplasia mainly involved the interdental papilla and gingival margin. Only a small number of those affected with severe gingival attachment; and violations of the disease also attached gingiva, interdental papilla and marginal gingival; ⑧ extent of drug-induced gingival hyperplasia is relatively light. Hyperplasia, covering up the crown about 1 / 3, and the disease often covered 2 / 3; ④ gingival hyperplasia large Lunding of chronic inflammation associated with more inflammatory cells observed histologically hyperplastic gingivitis and fibers were similar, but the disease is occasionally inflammatory cells. (2) fiber-type hyperplastic gingivitis, usually accompanied by inflammation, illness range of mainly anterior, violations of the interdental papilla and gingival margin. Gingival hyperplasia is generally not more than the tooth surface covered by the crown 1 / 3. Significant local incentives. No long-term medication history and family history. (3) is sometimes associated with gingival fibromatosis hairy psychosis, as part of a number of other congenital syndrome. Also occurred in the gums with a number of differentiated tumors. Treatment consists mainly of long gum surgery to restore appearance and function, some people advocate the use of the oblique cut-style flap operation. Cater to the gum removed, it can keep attached gingiva,
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, and shorten the healing process. The disease recur after surgery. Recurrence rate depends largely on the quality of oral health. To maintain good oral hygiene, you can not relapse or recurrence of slow 10 ~ 2O years. The disease is benign, although relapse even after surgery. Extraction, generally not considered. . II buildings. , ● J. fl do not, the hospital,
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