Strona glówna
•
FAQ
•
Szukaj
•
Użytkownicy
•
Grupy
•
Galerie
•
Rejestracja
•
Profil
•
Zaloguj się, by sprawdzić wiadomości
•
Zaloguj
Forum Forum MESA !! Strona Główna
->
Wisła Kraków
Napisz odpowiedź
Użytkownik
Temat
Treść wiadomości
Emotikony
Więcej Ikon
Kolor:
Domyślny
Ciemnoczerwony
Czerwony
Pomarańćzowy
Brązowy
Żółty
Zielony
Oliwkowy
Błękitny
Niebieski
Ciemnoniebieski
Purpurowy
Fioletowy
Biały
Czarny
Rozmiar:
Minimalny
Mały
Normalny
Duży
Ogromny
Zamknij Tagi
Opcje
HTML:
TAK
BBCode
:
TAK
Uśmieszki:
TAK
Wyłącz HTML w tym poście
Wyłącz BBCode w tym poście
Wyłącz Uśmieszki w tym poście
Kod potwierdzający: *
Wszystkie czasy w strefie EET (Europa)
Skocz do:
Wybierz forum
Nabór do ZESPÓŁ Forum MESA
----------------
Nabór
MESA - DRUŻYNY
----------------
GKS Bełchatów
Cracovia Kraków
Dyskobolia Grodzisk Wielkopolski
Górnik Zabrze
Jagiellonia Białystok
Korona Kielce
Lech Poznań
Legia Warszawa
ŁKS Łódź
Odra Wodzisław Śląski
Polonia Bytom
Ruch Chorzów
Wisła Kraków
Zagłębie Lubin
Zagłębie Sosnowiec
MESA - OGÓLNIE
----------------
Regulamin
Terminarz
Sędziowie
Wyniki
DLA KIBICA
----------------
Typer
Rozrywka
Sonda
INNE
----------------
Hydepark
Reklama
PARTNERZY
----------------
Parnerzy w reklamie i realizacji projektu !!
Przegląd tematu
Autor
Wiadomość
asbryobvrz
Wysłany: Pią 6:30, 25 Mar 2011
Temat postu: tory burch reva jen erq kvtx rwk
Cerebral salt wasting syndrome of
Death, the death rate 11.76%. 3 Discussion CSWS or SIADH is due to hypothalamic-pituitary damage caused by various reasons (adrenocorticotropichormone, ACrH) and (antidiuretichormone, ADH) abnormal secretion, so that the increase in renal water reabsorption, urinary sodium excretion increased, on the basis appear on the lower plasma osmolality and serum sodium [1】. CSWS is the danger of reduced plasma osmolality and serum sodium to promote water transport to the cells, resulting in intracellular edema, cerebral edema, in which the most dangerous [2】. It has been reported in 2Ah sharp decline in serum sodium to 120mmol / L or less up to 50% mortality due to cerebral edema is irreversible 【3】. 5 patients in this group, 2 patients died in critically ill patients. CSWS clinical diagnosis, the current gold standard for diagnosis is n】: serum sodium 80mmol/24h; urinary osmolality / plasma osmolality> 1. The key is to have a vigilance, I believe that if the general stability of post-operative patients,
tory burch reva
, 72h after surgery or after a sudden decline in progressive state of consciousness, no other reasons could explain, consider SIADH. Analysis of clinical manifestations can be found, who showed mild gastrointestinal symptoms and progressive fall in consciousness, and consciousness is manifested in patients with severe conditions to deteriorate rapidly. CSWS is the significance of early detection: the result of other causes brain edema and cerebral edema in the treatment of the existence of different points. General cerebral edema preferred hypertonic dehydration,
herve leger toronto
, but CSWS cerebral edema, is not only ineffective treatment for critically ill patients can increase the symptoms. Hypertonic dehydration due to further promote urinary sodium excretion of 25, leading to clinical symptoms. For mild cases of high dose furosemide have more significant effect, but no significant effect of critically ill patients, should adopt an integrated treatment. I also found that there are limitations restrict water intake: elderly patients with a longer time limit water intake (48 ~ 72h or more), while high doses dehydration or diuretics, can cause a significant amount of water exceeds intake of water, the blood concentration , blood viscosity increased, decreased effective circulating blood volume, induced heart, brain hypoperfusion, and adequate cerebral perfusion is a decisive factor influencing the prognosis. While limiting the intake of water can be too viscous airway secretions,
MBT schuhe preise
, cough, or aspiration is not easy,
tory burch outlet
, easy to induce lower respiratory tract infection,
herve leger outlet
, tracheotomy patients, especially for the disadvantaged. The author's experience is for mild cases: fluid in the appropriate amount of control on the basis of (15 (~ ml/24h so), make appropriate use of furosemide and sodium increased with the drug in plasma osmotic pressure, can achieve the desired effect; for critically ill patients: First, large doses of albumin (20 ~ 30g/24h) and furosemide (O.5 ~ 1.0/24h) control the cerebral edema, and actively make sodium. hypertonic salt water should be used (3% ~ 5 %), the serum sodium in a short time rose to 130mmol / L. hypertonic sodium current found in water treatment has a positive role in brain edema. Further also added ACTH (50 ~ 100U/24h), by negative feedback inhibition of ACTH ADH role. At the same time the serum sodium, urine sodium and urine osmolarity is to assess the efficacy of active surveillance to determine the gold standard for further treatment. 【
],[
fora.pl
- załóż własne forum dyskusyjne za darmo
Theme
FrayCan
created by
spleen
&
Download
Powered by
phpBB
© 2001, 2005 phpBB Group
Regulamin