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Losartan and captopril in spontaneously hypertensive rats of Myocardial Fibrosis Beneficial cardiac remodeling. In many pathological conditions,tory burch, the alternative pathway of AngII generation accounts for about 80% of ACEI can not suppress this part of AngII generation, and therefore not completely inhibited AngII generation, which may improve collagen in phenotype,tory burch shoes, increased collagen solubility, lower cross-linking of collagen and improve the role of MMPs-1 activity than losartan reasons. Blocking the renin-angiotensin system, regardless of the role of AngII at the receptor level or build one of the ways they were able to reduce the hydroxyproline content,mbt scarpe, improve collagen phenotype,mulberry bags, reduced collagen cross-linking, inhibition and reversal of AngII increased soluble mediated role in promoting myocardial fibrosis. However, to improve the collagen phenotype and decreased collagen cross-linking, soluble collagen in terms of improving effect of losartan is superior to captopril. The study also reflect the reversal of myocardial fibrosis is a long process, indicating the importance of long-term treatment of clinical hypertension. |
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