adidas scarpe ptg uzy uhav ibq

asbryobvrz · Wysłany: Nie 23:52, 06 Mar 2011

,[link widoczny dla zalogowanych]
Losartan and captopril in spontaneously hypertensive rats of Myocardial Fibrosis

Beneficial cardiac remodeling. In many pathological conditions,[link widoczny dla zalogowanych], the alternative pathway of AngII generation accounts for about 80% of ACEI can not suppress this part of AngII generation, and therefore not completely inhibited AngII generation, which may improve collagen in phenotype,[link widoczny dla zalogowanych], increased collagen solubility, lower cross-linking of collagen and improve the role of MMPs-1 activity than losartan reasons. Blocking the renin-angiotensin system, regardless of the role of AngII at the receptor level or build one of the ways they were able to reduce the hydroxyproline content,[link widoczny dla zalogowanych], improve collagen phenotype,[link widoczny dla zalogowanych], reduced collagen cross-linking, inhibition and reversal of AngII increased soluble mediated role in promoting myocardial fibrosis. However, to improve the collagen phenotype and decreased collagen cross-linking, soluble collagen in terms of improving effect of losartan is superior to captopril. The study also reflect the reversal of myocardial fibrosis is a long process, indicating the importance of long-term treatment of clinical hypertension.