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 Wysłany: Śro 1:31, 23 Mar 2011 |
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Non- enzymatic glycation inhibitor on the ultrastructure of the role of diabetic rats
Such as collagen and other proteins is a special vulnerability of the . Diabetes, myocardial interstitial ,[link widoczny dla zalogowanych], capillary wall collagen prone NEG,[link widoczny dla zalogowanych], formed almost . Generous in the myocardial interstitial and capillary wall deposition ,[link widoczny dla zalogowanych], there may be myocardial sclerosis, decreased compliance , microvascular basement membrane thickening , luminal narrowing , leading to cardiac insufficiency , cardiac function and further increase the damage. AG does not reduce blood sugar, but can suppress the increase of serum fructosamine , suggesting that AG not only inhibits the formation of AGEs advanced glycation products of early glycation products have a certain extent. AG intervention in this study,[link widoczny dla zalogowanych], diabetic rats treated microvascular and interstitial disease was significantly improved to some extent ,[link widoczny dla zalogowanych], suggesting that AG may be by blocking NEG, reducing AGEs in the myocardial interstitium and the microvascular deposition of diabetic cardiomyopathy has played an effective role . [
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